WB analysis of various samples using GTX12131 ABCG2 antibody. Lane 1 : rat liver tissue lysate at 50ug Lane 2 : rat spleen tissue lysate at 50ug Lane 3 : rat kidney tissue lysate at 50ug Lane 4 : mouse spleen tissue lysate at 50ug Dilution : 0.5 microg/mL
ABCG2 antibody
GTX12131
Overview
- SupplierGeneTex
- Product NameABCG2 antibody
- Delivery Days Customer9
- ApplicationsWestern Blot
- CertificationResearch Use Only
- ClonalityPolyclonal
- Concentration500 ug/ml
- ConjugateUnconjugated
- Gene ID26357
- Target nameAbcg2
- Target descriptionATP binding cassette subfamily G member 2 (Junior blood group)
- Target synonymsABC15; ABCP; AI428558; ATP-binding cassette sub-family G member 2; ATP-binding cassette, sub-family G (WHITE), member 2; Bc; BCRP; Bcrp1; breast cancer resistance protein 1 homolog; broad substrate specificity ATP-binding cassette transporter ABCG2; mitoxantrone resistance protein 1; MXR; MXR1; urate exporter
- HostRabbit
- IsotypeIgG
- Scientific DescriptionThe membrane-associated protein encoded by this gene is included in the superfamily of ATP-binding cassette (ABC) transporters. ABC proteins transport various molecules across extra- and intra-cellular membranes. ABC genes are divided into seven distinct subfamilies (ABC1, MDR/TAP, MRP, ALD, OABP, GCN20, White). This protein is a member of the White subfamily. Alternatively referred to as a breast cancer resistance protein, the human protein functions as a xenobiotic transporter which may play a major role in multi-drug resistance. This protein likely serves as a cellular defense mechanism in response to mitoxantrone and anthracycline exposure. [provided by RefSeq, Jul 2008]
- ReactivityMouse, Rat
- Storage Instruction2°C to 8°C,-20°C or -80°C
- UNSPSC12352203
References
- Probiotic supplements prevented oxonic acid-induced hyperuricemia and renal damage. Garcia-Arroyo FE et al., 2018, PLoS OneRead more
- ATP Binding Cassette Sub-family Member 2 (ABCG2) and Xenobiotic Exposure During Early Mouse Embryonic Stem Cell Differentiation. Rosen MB et al., 2018 Jan 15, Birth Defects ResRead more