![IHC-P analysis of human skeletal muscle tissue using GTX29465 sarcomeric alpha Actinin antibody [EA-53]. IHC-P analysis of human skeletal muscle tissue using GTX29465 sarcomeric alpha Actinin antibody [EA-53].](https://www.genetex.com/upload/website/prouct_img/normal/GTX29465/GTX29465_20191203_IHC-P_131_w_23060722_998.webp)
IHC-P analysis of human skeletal muscle tissue using GTX29465 sarcomeric alpha Actinin antibody [EA-53].
sarcomeric alpha Actinin antibody [EA-53]
GTX29465
ApplicationsImmunoFluorescence, ImmunoCytoChemistry, ImmunoHistoChemistry, ImmunoHistoChemistry Frozen, ImmunoHistoChemistry Paraffin
Product group Antibodies
ReactivityHuman, Mouse, Rabbit, Zebra Fish
TargetACTN3
Overview
- SupplierGeneTex
- Product Namesarcomeric alpha Actinin antibody [EA-53]
- Delivery Days Customer9
- Application Supplier NoteIHC-P: 1:20-1:30. *Optimal dilutions/concentrations should be determined by the researcher.Not tested in other applications.
- ApplicationsImmunoFluorescence, ImmunoCytoChemistry, ImmunoHistoChemistry, ImmunoHistoChemistry Frozen, ImmunoHistoChemistry Paraffin
- CertificationResearch Use Only
- ClonalityMonoclonal
- Clone IDEA-53
- ConjugateUnconjugated
- Gene ID100009544
- Target nameACTN3
- Target descriptionactinin alpha 3
- Target synonymsalpha-actinin-3, muscle alpha-actinin subunit
- HostMouse
- IsotypeIgG1
- ReactivityHuman, Mouse, Rabbit, Zebra Fish
- Storage Instruction2°C to 8°C
- UNSPSC12352203
References
- Gong R, Wang X, Li H, et al. Loss of m(6)A methyltransferase METTL3 promotes heart regeneration and repair after myocardial injury. Pharmacol Res. 2021,174:105845. doi: 10.1016/j.phrs.2021.105845Read this paper
- Bellmann J, Goswami RY, Girardo S, et al. A customizable microfluidic platform for medium-throughput modeling of neuromuscular circuits. Biomaterials. 2019,225:119537. doi: 10.1016/j.biomaterials.2019.119537Read this paper
- Williams AL, Khadka V, Tang M, et al. HIF1 mediates a switch in pyruvate kinase isoforms after myocardial infarction. Physiol Genomics. 2018,50(7):479-494. doi: 10.1152/physiolgenomics.00130.2017Read this paper
- Milan M, Pace V, Maiullari F, et al. Givinostat reduces adverse cardiac remodeling through regulating fibroblasts activation. Cell Death Dis. 2018,9(2):108. doi: 10.1038/s41419-017-0174-5Read this paper
- Zou J, Tran D, Baalbaki M, et al. An internal promoter underlies the difference in disease severity between N- and C-terminal truncation mutations of Titin in zebrafish. Elife. 2015,4:e09406. doi: 10.7554/eLife.09406Read this paper
- Burridge PW, Thompson S, Millrod MA, et al. A universal system for highly efficient cardiac differentiation of human induced pluripotent stem cells that eliminates interline variability. PLoS One. 2011,6(4):e18293. doi: 10.1371/journal.pone.0018293Read this paper
