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WB analysis of A-20 cell lysate using GTX85390 Slc35D1 antibody. Working concentration : (A) 1 and (B) 2 microg/ml
WB analysis of A-20 cell lysate using GTX85390 Slc35D1 antibody. Working concentration : (A) 1 and (B) 2 microg/ml
WB analysis of A-20 cell lysate using GTX85390 Slc35D1 antibody. Working concentration : (A) 1 and (B) 2 microg/ml

Slc35D1 antibody

GTX85390
GeneTex
ApplicationsWestern Blot, ELISA
DownloadDatasheet
Product group Antibodies
ReactivityHuman, Mouse, Rat
TargetSLC35D1
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Overview

  • Supplier
    GeneTex
  • Product Name
    Slc35D1 antibody
  • Delivery Days Customer
    9
  • Antibody Specificity
    This antibody is predicted to not cross-react with the highly homologous Slc35D2.
  • Application Supplier Note
    WB: 1 - 2 microg/mL. *Optimal dilutions/concentrations should be determined by the researcher.Not tested in other applications.
  • Applications
    Western Blot, ELISA
  • Certification
    Research Use Only
  • Clonality
    Polyclonal
  • Concentration
    1 mg/ml
  • Conjugate
    Unconjugated
  • Gene ID23169
  • Target name
    SLC35D1
  • Target description
    solute carrier family 35 member D1
  • Target synonyms
    SHNKND; solute carrier family 35 (UDP-GlcA/UDP-GalNAc transporter), member D1; solute carrier family 35 (UDP-glucuronic acid/UDP-N-acetylgalactosamine dual transporter), member D1; UDP-galactose transporter-related 7; UDP-galactose transporter-related protein 7; UDP-GlcA/UDP-GalNAc transporter; UDP-glucuronic acid/UDP-N-acetylgalactosamine transporter; UGTREL7
  • Host
    Rabbit
  • Isotype
    IgG
  • Protein IDQ9NTN3
  • Protein Name
    UDP-glucuronic acid/UDP-N-acetylgalactosamine transporter
  • Scientific Description
    The solute carrier family Slc35 consists of at least 17 proteins that act as nucleotide sugar transporters localized to the Golgi apparatus and endoplasmic reticulum. The role of the ER-resident Slc family member Slc35D1 is to transport both UDP-glucuronic acid and UDP-N-acetylgalactosamine. These molecules can serve as substrates for chondroitin sulfate biosynthesis and mice lacking the Slc35D1 gene developed a lethal form of skeletal dysplasia with severe shortening of limbs and facial structures. Examination of epiphyseal cartilage in these mice revealed a decreased proliferating zone with round chrondrocytes, scarce matrices, and reduced proteoglycan aggregates. Loss of function mutations in human Slc35D1 cause Schneckenbecken dysplasia, a severe skeletal dysplasia. This antibody is predicted to not cross-react with the highly homologous Slc35D2.
  • Reactivity
    Human, Mouse, Rat
  • Storage Instruction
    -20°C or -80°C,2°C to 8°C
  • UNSPSC
    12352203