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CRMP1 antibody

GTX114940
GeneTex
ApplicationsWestern Blot, ImmunoHistoChemistry, ImmunoHistoChemistry Paraffin
Product group Antibodies
TargetCRMP1
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Overview

  • Supplier
    GeneTex
  • Product Name
    CRMP1 antibody
  • Delivery Days Customer
    9
  • Application Supplier Note
    WB: 1:1000-1:10000. IHC-P: 1:100-1:1000. *Optimal dilutions/concentrations should be determined by the researcher.Not tested in other applications.
  • Applications
    Western Blot, ImmunoHistoChemistry, ImmunoHistoChemistry Paraffin
  • Certification
    Research Use Only
  • Clonality
    Polyclonal
  • Concentration
    0.77 mg/ml
  • Conjugate
    Unconjugated
  • Gene ID1400
  • Target name
    CRMP1
  • Target description
    collapsin response mediator protein 1
  • Target synonyms
    CRMP-1, DPYSL1, DRP-1, DRP1, ULIP-3, dihydropyrimidinase-related protein 1, dihydropyrimidinase-like 1, inactive dihydropyrimidinase, unc-33-like phosphoprotein 3
  • Host
    Rabbit
  • Isotype
    IgG
  • Protein IDQ14194
  • Protein Name
    Dihydropyrimidinase-related protein 1
  • Scientific Description
    This gene encodes a member of a family of cytosolic phosphoproteins expressed exclusively in the nervous system. The encoded protein is thought to be a part of the semaphorin signal transduction pathway implicated in semaphorin-induced growth cone collapse during neural development. Alternative splicing results in multiple transcript variants. [provided by RefSeq, Jul 2008]
  • Storage Instruction
    -20°C or -80°C,2°C to 8°C
  • UNSPSC
    12352203

References

  • Lin YF, Chen CA, Hsu FY, et al. Elevated Hippocampal CRMP5 Mediates Chronic Stress-Induced Cognitive Deficits by Disrupting Synaptic Plasticity, Hindering AMPAR Trafficking, and Triggering Cytokine Release. Int J Mol Sci. 2023,24(5). doi: 10.3390/ijms24054898
    Read this paper
  • Lin YS, Lin YF, Chen KC, et al. Collapsin response mediator protein 5 (CRMP5) causes social deficits and accelerates memory loss in an animal model of Alzheimer's disease. Neuropharmacology. 2019,157:107673. doi: 10.1016/j.neuropharm.2019.107673
    Read this paper