
WB analysis of human muscle extract using GTX11428 S100 alpha antibody.
S100 alpha antibody
GTX11428
ApplicationsImmunoFluorescence, ImmunoPrecipitation, Western Blot, ELISA, ImmunoCytoChemistry, ImmunoHistoChemistry, ImmunoHistoChemistry Paraffin
Product group Antibodies
ReactivityBovine, Human, Mouse, Porcine, Rat
TargetS100A1
Overview
- SupplierGeneTex
- Product NameS100 alpha antibody
- Delivery Days Customer9
- Application Supplier NoteWB: 0.1 microg/ml. ICC/IF: 1:50-1:200. IHC-P: 1 microg/ml. IP: 5 microg/ml. *Optimal dilutions/concentrations should be determined by the researcher.Not tested in other applications.
- ApplicationsImmunoFluorescence, ImmunoPrecipitation, Western Blot, ELISA, ImmunoCytoChemistry, ImmunoHistoChemistry, ImmunoHistoChemistry Paraffin
- CertificationResearch Use Only
- ClonalityPolyclonal
- ConjugateUnconjugated
- Gene ID6271
- Target nameS100A1
- Target descriptionS100 calcium binding protein A1
- Target synonymsS100, S100-alpha, S100A, protein S100-A1, S-100 protein alpha chain, S-100 protein subunit alpha, S100 alpha, S100 protein, alpha polypeptide
- HostRabbit
- IsotypeIgG
- Protein IDP23297
- Protein NameProtein S100-A1
- Scientific DescriptionThe protein encoded by this gene is a member of the S100 family of proteins containing 2 EF-hand calcium-binding motifs. S100 proteins are localized in the cytoplasm and/or nucleus of a wide range of cells, and involved in the regulation of a number of cellular processes such as cell cycle progression and differentiation. S100 genes include at least 13 members which are located as a cluster on chromosome 1q21. This protein may function in stimulation of Ca2+-induced Ca2+ release, inhibition of microtubule assembly, and inhibition of protein kinase C-mediated phosphorylation. Reduced expression of this protein has been implicated in cardiomyopathies. [provided by RefSeq, Jul 2008]
- ReactivityBovine, Human, Mouse, Porcine, Rat
- Storage Instruction-20°C or -80°C,2°C to 8°C
- UNSPSC12352203
References
- Desjardins JF, Pourdjabbar A, Quan A, et al. Lack of S100A1 in mice confers a gender-dependent hypertensive phenotype and increased mortality after myocardial infarction. Am J Physiol Heart Circ Physiol. 2009,296(5):H1457-65. doi: 10.1152/ajpheart.00088.2008Read this paper




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