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IHC-P analysis of human heart tissue using GTX47128 ALAD antibody at 1:100. Left to right:DAPI, ALAD, Merge. Heat-induced antigen retrieval method utilizing Sodium Citrate buffer was performed.
IHC-P analysis of human heart tissue using GTX47128 ALAD antibody at 1:100. Left to right:DAPI, ALAD, Merge. Heat-induced antigen retrieval method utilizing Sodium Citrate buffer was performed.
IHC-P analysis of human heart tissue using GTX47128 ALAD antibody at 1:100. Left to right:DAPI, ALAD, Merge. Heat-induced antigen retrieval method utilizing Sodium Citrate buffer was performed.

ALAD antibody, Internal

GTX47128
GeneTex
ApplicationsWestern Blot, ImmunoHistoChemistry, ImmunoHistoChemistry Paraffin
DownloadDatasheet
Product group Antibodies
ReactivityHuman
TargetALAD
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Overview

  • Supplier
    GeneTex
  • Product Name
    ALAD antibody, Internal
  • Delivery Days Customer
    9
  • Application Supplier Note
    WB: 0.2-2.5 ug/ml. IHC-P: 2-10 ug/ml. *Optimal dilutions/concentrations should be determined by the researcher.Not tested in other applications.
  • Applications
    Western Blot, ImmunoHistoChemistry, ImmunoHistoChemistry Paraffin
  • Certification
    Research Use Only
  • Clonality
    Polyclonal
  • Concentration
    0.5-1 mg/ml
  • Conjugate
    Unconjugated
  • Gene ID210
  • Target name
    ALAD
  • Target description
    aminolevulinate dehydratase
  • Target synonyms
    ALADH; aminolevulinate, delta-, dehydratase; delta-aminolevulinic acid dehydratase; PBGS; porphobilinogen synthase; testicular tissue protein Li 95
  • Host
    Rabbit
  • Isotype
    IgG
  • Protein IDP13716
  • Protein Name
    Delta-aminolevulinic acid dehydratase
  • Scientific Description
    The ALAD enzyme is composed of 8 identical subunits and catalyzes the condensation of 2 molecules of delta-aminolevulinate to form porphobilinogen (a precursor of heme, cytochromes and other hemoproteins). ALAD catalyzes the second step in the porphyrin and heme biosynthetic pathway; zinc is essential for enzymatic activity. ALAD enzymatic activity is inhibited by lead and a defect in the ALAD structural gene can cause increased sensitivity to lead poisoning and acute hepatic porphyria. Alternative splicing of this gene results in multiple transcript variants encoding different isoforms. [provided by RefSeq, Dec 2015]
  • Reactivity
    Human
  • Storage Instruction
    -20°C or -80°C,2°C to 8°C
  • UNSPSC
    12352203