anti-FGF-23 (human), mAb (FG322-3)
AG-20A-0073
ApplicationsWestern Blot, ELISA, ImmunoHistoChemistry
Product group Antibodies
ReactivityHuman
TargetFGF23
Overview
- SupplierAdipoGen Life Sciences
- Product Nameanti-FGF-23 (human), mAb (FG322-3)
- Delivery Days Customer10
- Antibody SpecificityRecognizes human FGF-23. Does not cross-react with mouse FGF-23.
- ApplicationsWestern Blot, ELISA, ImmunoHistoChemistry
- CertificationResearch Use Only
- ClonalityMonoclonal
- Clone IDFG322-3
- Concentration1 mg/ml
- FormulationLiquid
- Gene ID8074
- Target nameFGF23
- Target descriptionfibroblast growth factor 23
- Target synonymsADHR; FGFN; fibroblast growth factor 23; HFTC2; HPDR2; HYPF; phosphatonin; PHPTC; tumor-derived hypophosphatemia inducing factor
- HostMouse
- IsotypeIgG1
- Protein IDQ9GZV9
- Protein NameFibroblast growth factor 23
- Scientific DescriptionFGF-23 is a regulator of phosphate homeostasis. It upregulates EGR1 expression in the presence of KLBy. Acts directly on the parathyroid to decrease PTH secretion. Regulates the vitamin-D metabolism. Negatively regulates osteoblast differentiation and matrix mineralization. Defects in FGF-23 are the cause of autosomal dominant hypophosphataemic rickets (ADHR) and of hyperphosphatemic familial tumoral calcinosis (HFTC). - Monoclonal Antibody. Recognizes human FGF-23. Does not cross-react with mouse FGF-23. Isotype: Mouse IgG1kappa. Clone: FG322-3. Applications: ELISA, IHC, WB. Liquid. 0.2microm-filtered solution in PBS, pH 7.4. Contains no preservatives. FGF-23 is a regulator of phosphate homeostasis. It upregulates EGR1 expression in the presence of KLBy. Acts directly on the parathyroid to decrease PTH secretion. Regulates the vitamin-D metabolism. Negatively regulates osteoblast differentiation and matrix mineralization. Defects in FGF-23 are the cause of autosomal dominant hypophosphataemic rickets (ADHR) and of hyperphosphatemic familial tumoral calcinosis (HFTC).
- ReactivityHuman
- Storage Instruction-20°C,2°C to 8°C
- UNSPSC12352203