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ProductsBack/E2F3 antibody [PG30]

WB analysis of 293T cell lysates using GTX11843 E2F3 antibody [PG30]. Dilution : Lane1 : 4 microg/mL Lane2 : 8 microg/mL

WB analysis of 293T cell lysates using GTX11843 E2F3 antibody [PG30]. Dilution : Lane1 : 4 microg/mL Lane2 : 8 microg/mL

WB analysis of 293T cell lysates using GTX11843 E2F3 antibody [PG30]. Dilution : Lane1 : 4 microg/mL Lane2 : 8 microg/mL

E2F3 antibody [PG30]

GTX11843

ApplicationsImmunoPrecipitation, Western Blot

Product group Antibodies

ReactivityHuman

TargetE2F3

Overview

Supplier
GeneTex
Product Name
E2F3 antibody [PG30]
Delivery Days Customer
9
Application Supplier Note
WB: 4-8 microg/ml. *Optimal dilutions/concentrations should be determined by the researcher.Not tested in other applications.
Applications
ImmunoPrecipitation, Western Blot
Certification
Research Use Only
Clonality
Monoclonal
Clone ID
PG30
Concentration
2 mg/ml
Conjugate
Unconjugated
Gene ID1871
Target name
E2F3
Target description
E2F transcription factor 3
Target synonyms
E2F-3, transcription factor E2F3
Host
Mouse
Isotype
IgG2a
Protein IDO00716
Protein Name
Transcription factor E2F3
Scientific Description
E2F transcription factors are functionally regulated by binding to Rb p110, p107, and p130. E2F 3 is regulated by complex formation with Rb p110. E2F family members bind DNA as heterodimers with members of the DP family of polypeptides.
Reactivity
Human
Storage Instruction
-20°C or -80°C,2°C to 8°C
UNSPSC
12352203

References

Simoneschi D, Rona G, Zhou N, et al. CRL4(AMBRA1) is a master regulator of D-type cyclins. Nature. 2021,592(7856):789-793. doi: 10.1038/s41586-021-03445-y
Read this paper
Giro-Perafita A, Luo L, Khodadadi-Jamayran A, et al. LncRNA RP11-19E11 is an E2F1 target required for proliferation and survival of basal breast cancer. NPJ Breast Cancer. 2020,6:1. doi: 10.1038/s41523-019-0144-4
Read this paper
Clijsters L, Hoencamp C, Calis JJA, et al. Cyclin F Controls Cell-Cycle Transcriptional Outputs by Directing the Degradation of the Three Activator E2Fs. Mol Cell. 2019,74(6):1264-1277.e7. doi: 10.1016/j.molcel.2019.04.010
Read this paper

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