InVivoMAb anti-mouse CD11b

Neutrophil myeloperoxidase regulates T-cell-driven tissue inflammation in mice by inhibiting dendritic cell function.

The glycan-rich outer layer of the cell wall of Mycobacterium tuberculosis acts as an antiphagocytic capsule limiting the association of the bacterium with macrophages.

Rapid recruitment of inflammatory monocytes is independent of neutrophil migration.

Enhancement of G-CSF-induced stem cell mobilization by antibodies against the beta 2 integrins LFA-1 and Mac-1.

IFN-gamma action in the media of the great elastic arteries, a novel immunoprivileged site.

Complement receptor type 3 plays an important role in development of protective immunity to primary and secondary Corynebacterium pseudotuberculosis infection in mice.

Complement receptor 3 of macrophages is associated with galectin-1-like protein.

L1 antibodies block lymph node fibroblastic reticular matrix remodeling in vivo.

Expression and function of the type 3 complement receptor in tissues of the developing mouse.

Mouse CD23 regulates monocyte activation through an interaction with the adhesion molecule CD11b/CD18.