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WB analysis of samples using GTX24873 IRS1 (phospho Tyr896) antibody. The data show that only the immunogen phosphopeptide blocks the signal, demonstrating the specificity of the antibody.
WB analysis of samples using GTX24873 IRS1 (phospho Tyr896) antibody. The data show that only the immunogen phosphopeptide blocks the signal, demonstrating the specificity of the antibody.
WB analysis of samples using GTX24873 IRS1 (phospho Tyr896) antibody. The data show that only the immunogen phosphopeptide blocks the signal, demonstrating the specificity of the antibody.

IRS1 (phospho Tyr896) antibody

GTX24873
GeneTex
ApplicationsWestern Blot, ImmunoHistoChemistry, ImmunoHistoChemistry Paraffin
Product group Antibodies
ReactivityHuman, Mouse
TargetIRS1
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Overview

  • Supplier
    GeneTex
  • Product Name
    IRS1 (phospho Tyr896) antibody
  • Delivery Days Customer
    9
  • Application Supplier Note
    IHC-P: 1:10-1:100. *Optimal dilutions/concentrations should be determined by the researcher.Not tested in other applications.
  • Applications
    Western Blot, ImmunoHistoChemistry, ImmunoHistoChemistry Paraffin
  • Certification
    Research Use Only
  • Clonality
    Polyclonal
  • Conjugate
    Unconjugated
  • Gene ID3667
  • Target name
    IRS1
  • Target description
    insulin receptor substrate 1
  • Target synonyms
    HIRS-1, insulin receptor substrate 1
  • Host
    Rabbit
  • Isotype
    IgG
  • Protein IDP35568
  • Protein Name
    Insulin receptor substrate 1
  • Scientific Description
    This gene encodes a protein which is phosphorylated by insulin receptor tyrosine kinase. Mutations in this gene are associated with type II diabetes and susceptibility to insulin resistance. [provided by RefSeq, Nov 2009]
  • Reactivity
    Human, Mouse
  • Storage Instruction
    -20°C or -80°C,2°C to 8°C
  • UNSPSC
    12352203

References

  • Renna LV, Bosè F, Iachettini S, et al. Receptor and post-receptor abnormalities contribute to insulin resistance in myotonic dystrophy type 1 and type 2 skeletal muscle. PLoS One. 2017,12(9):e0184987. doi: 10.1371/journal.pone.0184987
    Read this paper

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