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TOMM20 antibody

GTX133756
GeneTex
ApplicationsImmunoFluorescence, Western Blot, ImmunoCytoChemistry, ImmunoHistoChemistry, ImmunoHistoChemistry Paraffin
Product group Antibodies
TargetTOMM20
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Overview

  • Supplier
    GeneTex
  • Product Name
    TOMM20 antibody
  • Delivery Days Customer
    9
  • Application Supplier Note
    WB: 1:500-1:3000. IHC-P: 1:100-1:1000. *Optimal dilutions/concentrations should be determined by the researcher.Not tested in other applications.
  • Applications
    ImmunoFluorescence, Western Blot, ImmunoCytoChemistry, ImmunoHistoChemistry, ImmunoHistoChemistry Paraffin
  • Certification
    Research Use Only
  • Clonality
    Polyclonal
  • Concentration
    0.21 mg/ml
  • Conjugate
    Unconjugated
  • Gene ID9804
  • Target name
    TOMM20
  • Target description
    translocase of outer mitochondrial membrane 20
  • Target synonyms
    MAS20, MOM19, TOM20, mitochondrial import receptor subunit TOM20 homolog, mitochondrial 20 kDa outer membrane protein, outer mitochondrial membrane receptor Tom20, translocase of outer mitochondrial membrane 20 homolog type II
  • Host
    Rabbit
  • Isotype
    IgG
  • Protein IDQ15388
  • Protein Name
    Mitochondrial import receptor subunit TOM20 homolog
  • Scientific Description
    Central component of the receptor complex responsible for the recognition and translocation of cytosolically synthesized mitochondrial preproteins. Together with TOM22 functions as the transit peptide receptor at the surface of the mitochondrion outer membrane and facilitates the movement of preproteins into the TOM40 translocation pore.
  • Storage Instruction
    -20°C or -80°C,2°C to 8°C
  • UNSPSC
    12352203

References

  • Chen KC, Chen CR, Chen CY, et al. Bicalutamide Exhibits Potential to Damage Kidney via Destroying Complex I and Affecting Mitochondrial Dynamics. J Clin Med. 2021,11(1). doi: 10.3390/jcm11010135
    Read this paper
  • Bosc C, Broin N, Fanjul M, et al. Autophagy regulates fatty acid availability for oxidative phosphorylation through mitochondria-endoplasmic reticulum contact sites. Nat Commun. 2020,11(1):4056. doi: 10.1038/s41467-020-17882-2
    Read this paper
  • Keller G, Binyamin O, Frid K, et al. Mitochondrial dysfunction in preclinical genetic prion disease: A target for preventive treatment? Neurobiol Dis. 2019,124:57-66. doi: 10.1016/j.nbd.2018.11.003
    Read this paper