WB analysis of various sample lysates using GTX32434 alpha 2 Macroglobulin antibody. Dilution : 1:500 Loading : 25microg per lane
WB analysis of various sample lysates using GTX32434 alpha 2 Macroglobulin antibody. Dilution : 1:500 Loading : 25microg per lane
alpha 2 Macroglobulin antibody
GTX32434
ApplicationsImmunoFluorescence, Western Blot, ImmunoCytoChemistry
Product group Antibodies
TargetA2M
Overview
- SupplierGeneTex
- Product Namealpha 2 Macroglobulin antibody
- Delivery Days Customer9
- Application Supplier NoteWB: 1:500 - 1:2000. ICC/IF: 1:50 - 1:200. *Optimal dilutions/concentrations should be determined by the researcher.Not tested in other applications.
- ApplicationsImmunoFluorescence, Western Blot, ImmunoCytoChemistry
- CertificationResearch Use Only
- ClonalityPolyclonal
- ConjugateUnconjugated
- Gene ID2
- Target nameA2M
- Target descriptionalpha-2-macroglobulin
- Target synonymsA2MD; alpha-2-M; alpha-2-macroglobulin; C3 and PZP-like alpha-2-macroglobulin domain-containing protein 5; CPAMD5; FWP007; S863-7
- HostRabbit
- IsotypeIgG
- Protein IDP01023
- Protein NameAlpha-2-macroglobulin
- Scientific DescriptionThe protein encoded by this gene is a protease inhibitor and cytokine transporter. It uses a bait-and-trap mechanism to inhibit a broad spectrum of proteases, including trypsin, thrombin and collagenase. It can also inhibit inflammatory cytokines, and it thus disrupts inflammatory cascades. Mutations in this gene are a cause of alpha-2-macroglobulin deficiency. This gene is implicated in Alzheimers disease (AD) due to its ability to mediate the clearance and degradation of A-beta, the major component of beta-amyloid deposits. A related pseudogene, which is also located on the p arm of chromosome 12, has been identified. [provided by RefSeq, Nov 2016]
- Storage Instruction-20°C or -80°C,2°C to 8°C
- UNSPSC12352203
References
- Combined proteomic and metabolomic analyses of cerebrospinal fluid from mice with ischemic stroke reveals the effects of a Buyang Huanwu decoction in neurodegenerative disease. Hsu WH et al., 2019, PLoS OneRead more
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