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ProductsBack/FGF-23 (mouse):Fc (human) (rec.)
ERK and FRS2alpha phosphorylation induced by FGF-23 in Klotho expressing cells. Klotho expressing HEK 293EBNA cells were serum starved for 16hr and then stimulated with mFGF-23-Fc (Prod. No. AG-40A-0128), and FGF-b (100ng/ml) (positive control) f
ERK and FRS2alpha phosphorylation induced by FGF-23 in Klotho expressing cells. Klotho expressing HEK 293EBNA cells were serum starved for 16hr and then stimulated with mFGF-23-Fc (Prod. No. AG-40A-0128), and FGF-b (100ng/ml) (positive control) f
ERK and FRS2alpha phosphorylation induced by FGF-23 in Klotho expressing cells. Klotho expressing HEK 293EBNA cells were serum starved for 16hr and then stimulated with mFGF-23-Fc (Prod. No. AG-40A-0128), and FGF-b (100ng/ml) (positive control) f

FGF-23 (mouse):Fc (human) (rec.)

Research Use Only
AG-40A-0128Y
AdipoGen Life Sciences
Protein IDQ9EPC2
DownloadMSDS
Product group Proteins / Signaling Molecules
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Overview

  • Supplier
    AdipoGen Life Sciences
  • Product Name
    FGF-23 (mouse):Fc (human) (rec.)
  • Delivery Days Customer
    10
  • Certification
    Research Use Only
  • Concentration
    0.1 mg/ml
  • Estimated Purity
    >90%
  • Protein IDQ9EPC2
  • Protein Name
    Fibroblast growth factor 23
  • Scientific Description
    FGF-23 (Fibroblast growth factor 23) is a regulator of phosphate homeostasis. It upregulates EGR1 expression in the presence of KLBy. Acts directly on the parathyroid to decrease PTH secretion. Regulates the vitamin-D metabolism. Negatively regulates osteoblast differentiation and matrix mineralization. Defects in FGF-23 are the cause of autosomal dominant hypophosphataemic rickets (ADHR) and of hyperphosphatemic familial tumoral calcinosis (HFTC). - Recombinant protein. Mouse FGF-23 (aa25-251) is fused at the C-terminus to the Fc portion of human IgG1. Activates ERK and FRS2alpha phosphorylation in Klotho expressing cells. Source/Host: HEK 293 cells. Purity: >90% (SDS-PAGE). Lyophilized. Contains PBS. FGF-23 (Fibroblast growth factor 23) is a regulator of phosphate homeostasis. It upregulates EGR1 expression in the presence of KLBy. Acts directly on the parathyroid to decrease PTH secretion. Regulates the vitamin-D metabolism. Negatively regulates osteoblast differentiation and matrix mineralization. Defects in FGF-23 are the cause of autosomal dominant hypophosphataemic rickets (ADHR) and of hyperphosphatemic familial tumoral calcinosis (HFTC).
  • Storage Instruction
    -20°C,2°C to 8°C
  • UNSPSC
    12352202