HDAC1 antibody
GTX100513
ApplicationsImmunoFluorescence, ImmunoPrecipitation, Western Blot, ChIP Chromatin ImmunoPrecipitation, ImmunoCytoChemistry, ImmunoHistoChemistry, ImmunoHistoChemistry Frozen, ImmunoHistoChemistry Paraffin
Product group Antibodies
TargetHDAC1
Overview
- SupplierGeneTex
- Product NameHDAC1 antibody
- Delivery Days Customer9
- Application Supplier NoteWB: 1:500-1:3000. ICC/IF: 1:100-1:1000. IHC-P: 1:100-1:1000. IP: 1:100-1:500. *Optimal dilutions/concentrations should be determined by the researcher.Not tested in other applications.
- ApplicationsImmunoFluorescence, ImmunoPrecipitation, Western Blot, ChIP Chromatin ImmunoPrecipitation, ImmunoCytoChemistry, ImmunoHistoChemistry, ImmunoHistoChemistry Frozen, ImmunoHistoChemistry Paraffin
- CertificationResearch Use Only
- ClonalityPolyclonal
- Concentration1 mg/ml
- ConjugateUnconjugated
- Gene ID3065
- Target nameHDAC1
- Target descriptionhistone deacetylase 1
- Target synonymsGON-10, HD1, KDAC1, RPD3, RPD3L1, histone deacetylase 1, protein deacetylase HDAC1, protein deacylase HDAC1, protein decrotonylase HDAC1, reduced potassium dependency, yeast homolog-like 1
- HostRabbit
- IsotypeIgG
- Protein IDQ13547
- Protein NameHistone deacetylase 1
- Scientific DescriptionHistone acetylation and deacetylation, catalyzed by multisubunit complexes, play a key role in the regulation of eukaryotic gene expression. The protein encoded by this gene belongs to the histone deacetylase/acuc/apha family and is a component of the histone deacetylase complex. It also interacts with retinoblastoma tumor-suppressor protein and this complex is a key element in the control of cell proliferation and differentiation. Together with metastasis-associated protein-2, it deacetylates p53 and modulates its effect on cell growth and apoptosis. [provided by RefSeq]
- Storage Instruction-20°C or -80°C,2°C to 8°C
- UNSPSC12352203
References
- Xu Z, Liu S, Feng C, et al. Acetylation of Checkpoint suppressor 1 enhances its stability and promotes the progression of triple-negative breast cancer. Cell Death Discov. 2022,8(1):474. doi: 10.1038/s41420-022-01269-xRead this paper
- Clauß O, Schäker-Hübner L, Wenzel B, et al. Development and Biological Evaluation of the First Highly Potent and Specific Benzamide-Based Radiotracer [(18)F]BA3 for Imaging of Histone Deacetylases 1 and 2 in Brain. Pharmaceuticals (Basel). 2022,15(3). doi: 10.3390/ph15030324Read this paper
- Lin HY, Wu HJ, Chen SY, et al. Epigenetic therapy combination of UNC0638 and CI-994 suppresses breast cancer via epigenetic remodeling of BIRC5 and GADD45A. Biomed Pharmacother. 2022,145:112431. doi: 10.1016/j.biopha.2021.112431Read this paper
- Abdollahi S, Dehghanian SZ, Hung LY, et al. Deciphering genes associated with diffuse large B-cell lymphoma with lymphomatous effusions: A mutational accumulation scoring approach. Biomark Res. 2021,9(1):74. doi: 10.1186/s40364-021-00330-8Read this paper
- Yuan H, Suzuki S, Hirata-Tsuchiya S, et al. PPARγ-Induced Global H3K27 Acetylation Maintains Osteo/Cementogenic Abilities of Periodontal Ligament Fibroblasts. Int J Mol Sci. 2021,22(16). doi: 10.3390/ijms22168646Read this paper
- Chen K, Zeng J, Sun Y, et al. Junction plakoglobin regulates and destabilizes HIF2α to inhibit tumorigenesis of renal cell carcinoma. Cancer Commun (Lond). 2021,41(4):316-332. doi: 10.1002/cac2.12142Read this paper
- Hu TM, Chung HS, Ping LY, et al. Differential Expression of Multiple Disease-Related Protein Groups Induced by Valproic Acid in Human SH-SY5Y Neuroblastoma Cells. Brain Sci. 2020,10(8). doi: 10.3390/brainsci10080545Read this paper
- Ochiai H, Hayashi T, Umeda M, et al. Genome-wide kinetic properties of transcriptional bursting in mouse embryonic stem cells. Sci Adv. 2020,6(25):eaaz6699. doi: 10.1126/sciadv.aaz6699Read this paper
- Lin YC, Wang JC, Wu MS, et al. Nifedipine Exacerbates Lipogenesis in the Kidney via KIM-1, CD36, and SREBP Upregulation: Implications from an Animal Model for Human Study. Int J Mol Sci. 2020,21(12). doi: 10.3390/ijms21124359Read this paper
- Deng M, Yang S, Ji Y, et al. Overexpression of peptidase inhibitor 16 attenuates angiotensin II-induced cardiac fibrosis via regulating HDAC1 of cardiac fibroblasts. J Cell Mol Med. 2020,24(9):5249-5259. doi: 10.1111/jcmm.15178Read this paper
Datasheet
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